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smc컨텐츠

연구업적물 : 1999 ~ 2010.6월말 현재 원내의료진 SCI(E)연구업적입니다.

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  • 연구업적물
    15-deoxyspergualin prevents mucosal injury by inhibiting production of TNF-alpha and down-regulating expression of MD-1 in a murine model of TNBS-induced colitis  

    [김성주 , INT IMMUNOPHARMACOL , 2007 ;7 (8) : 1003 - 1012]

  • 연구업적물
    15-deoxyspergualin prevents mucosal injury by inhibiting production of TNF-alpha and down-regulating expression of MD-1 in a murine model of TNBS-induced colitis  

    [조재원 , INT IMMUNOPHARMACOL , 2007 ;7 (8) : 1003 - 1012]

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    15-deoxyspergualin prevents mucosal injury by inhibiting production of TNF-alpha and down-regulating expression of MD-1 in a murine model of TNBS-induced colitis  

    [권준혁 , INT IMMUNOPHARMACOL , 2007 ;7 (8) : 1003 - 1012]

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    16-Cyclopentadienyl tricarbonyl Tc-99m 16-oxo-hexadecanoic acid: Synthesis and evaluation of fatty acid metabolism in mouse myocardium  

    [이경한 , J MED CHEM , 2008 ;51 (12) : 3630 - 3634]

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    16-Cyclopentadienyl tricarbonyl Tc-99m 16-oxo-hexadecanoic acid: Synthesis and evaluation of fatty acid metabolism in mouse myocardium  

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    17-[4-(2-[F-18]Fluoroethyl)-1H-1,2,3-triazol-1-yl]-6-thia-heptadecanoic Acid: A Potential Radiotracer for the Evaluation of Myocardial Fatty Acid Metabolism  

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  • 연구업적물
    17-[4-(2-[F-18]Fluoroethyl)-1H-1,2,3-triazol-1-yl]-6-thia-heptadecanoic Acid: A Potential Radiotracer for the Evaluation of Myocardial Fatty Acid Metabolism  

    [김병태 , BIOCONJUGATE CHEM , 2009 ;20 (6) : 1139 - 1145]

  • 연구업적물
    17-[4-(2-[F-18]Fluoroethyl)-1H-1,2,3-triazol-1-yl]-6-thia-heptadecanoic Acid: A Potential Radiotracer for the Evaluation of Myocardial Fatty Acid Metabolism  

    [최준영 , BIOCONJUGATE CHEM , 2009 ;20 (6) : 1139 - 1145]

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    17 beta-estradiol inhibits proliferation of cultured vascular smooth muscle cells induced by lysophosphatidylcholine via a nongenomic antioxidant mechanism  

    [이제호 , MENOPAUSE , 2001 ;8 (1) : 58 - 64]

  • 연구업적물
    17 beta-estradiol inhibits proliferation of cultured vascular smooth muscle cells induced by lysophosphatidylcholine via a nongenomic antioxidant mechanism  

    [김덕경 , MENOPAUSE , 2001 ;8 (1) : 58 - 64]

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